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D., a postdoctoral researcher in the laboratory of CSHL Professor David Tuveson, M. D., looked closely at cells sampled from 308 people with pancreatic cancer, one of the most lethal malignancies, with a 5-year survival rate of only 8%. This enabled Feigin and colleagues including computational biologist Tyler Garvin, Ph.

D., formerly of Adjunct Associate Professor Michael Schatz's lab, to focus narrowly on genome segments called gene promoters.

"Promoters are important in determining when specific genes are turned on and off," says Feigin, "and I became interested in figuring out whether mutations within promoters -- as opposed to within the genes they regulate -consistently affects the way cancers develop and sustain themselves." The team "looked all across the genome," Feigin says, "and, interestingly, while we did find mutations in promoters, we never found clusters of these mutations near any of the genes that prior research had already told us were typically mutated in pancreatic cancer." Genes called KRAS and p53 are mutated in the majority of pancreas cancer cells, for example.

But mutations in promoters sifted out of mountains of data by the team's novel mathematical formula, or algorithm, called GECCO, lay in genes never before implicated in pancreatic cancer.

This year’s contest for teen journalists, now in its eighth year, attracted astonishing work from hundreds of students around the globe. The top winners come away with amazing crystal trophies and every winner receives a custom-made certificate. Read entire article » By Noah Adelsberger Junior Reporter Youth Journalism International NEWARK, Ohio, U. Computer Science and Engineering student Megan Knox was inclass when someone warned the classroom about an emergency alert text they had received about an active shooter. Read entire article » Kaishi Lee Youth Journalism International SINGAPORE — September 11, 2001. America’s fall from grace has stirred up much antipathy and the terrorist attack has touched a universal nerve. Today is the anniversary of my grandfather’s death, and the events that happened today did not help at all.lie adjacent to, but not within, the sequences of the genes that they regulate.Therefore, promoters are "invisible" when only the exomes of cells are sequenced, as has been commonplace in cancer genetics research.If cancer is a disease precipitated by changes in genes, after all, we need to know lots about how and when different genes change in the many distinctive subtypes of cancer.But a new wave of research, exemplified by a study published in Nature Genetics by a team at Cold Spring Harbor Laboratory (CSHL), is significantly improving our ability to target cancer cells by studying "the other 98%" of DNA in human chromosomes, sometimes called the genome's "dark matter." Research led by Michael Feigin, Ph.

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